Myocardial fibrosis and curcumin

Abstract: Curcumin is known to improve cardiac function by balancing degradation and synthesis of collagens after myocardial infarction. Read this original research and sign up to receive Drug Design, Development and Therapy journal here: https://www.dovepress.com/articles.php?article_id=24575
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Figure 1 Representative tracing of pulse signals by inflating the tail cuff with simultaneous monitoring of the cuff pressure.

Attenuation of myocardial fibrosis with curcumin is mediated by modula

Figure 2 Detection of the AT1 receptor during Ang II infusion.

Figure 2 Detection of the receptor during Ang II infusion.

Figure 3 Detection of the AT2 receptor during Ang II infusion.

Figure 3 Detection of the receptor during Ang II infusion.

Figure 4 Macrophage accumulation during Ang II infusion.

Attenuation of myocardial fibrosis with curcumin is mediated by modula

Figure 5 Proliferation of fibroblasts and expression of TGFβ1 during Ang II infusion.

Figure 5 Proliferation of fibroblasts and expression of during Ang II infusion.

Figure 6 Phosphorylation of Smad2/3 during Ang II infusion.

Figure 6 Phosphorylation of during Ang II infusion.

Figure 7 Expression of collagen type I and fibrotic tissue formation during Ang II infusion.

Attenuation of myocardial fibrosis with curcumin is mediated by modula

Figure 8 Expression of ACE 2 during Ang II infusion.

Attenuation of myocardial fibrosis with curcumin is mediated by modula

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